seryi7 wrote:I have tried tongkat ali but it didn't work. I don't want to use nettles because I have read that it block 5 alpha reductase. About proviron, I haven't used it because I think it can reduce my testosterone production. I would like to use danazol but as in the case of proviron it can shut down hpta. If I take danazol the drop in shbg levels is permanent?
If you use the nettle {HP; "High Potency"} EXTRACT then you won't run into those Issues..statistically, it's more likely to loosen up the DHT from SHBG thus increasing free T & DHT. It's mechanism is less clear than some others but it is reported to work for libido issues.
Proviron is VERY VERY *Minimally* Suppressive - it also has anti-E effects and aromtase inhibition remains a very good way to suppress SHBG as well..in this case, Adex (Arimidex, Anastrzole) is most fit and it doesn't bottom out E2-levels like Letro.
So you don't run into the joint / bone issues with Adex .
Here's a good write-up...
http://area1255.blogspot.com/2015/02/natural-vs-pharmaceutical-aromatase.htmlAlso check out these links.
http://www.ncbi.nlm.nih.gov/pubmed/2892728http://www.steroidology.com/proviron-all-you-need-to-know/http://united-muscle.com/archive/index.php/t-2158.htmlhttps://forums.t-nation.com/t/experiences-with-proviron-dht/142618Naps Proviron is pretty decent price if you wanna take a look.
https://www.napsgear.org/product_info.php?ref=2522&products_id=120
https://www.napsgear.org/product_info.php?ref=2522&products_id=6877Naps arimidex for future Reference..
https://www.napsgear.org/product_info.php?ref=2522&products_id=7083
Int J Gynaecol Obstet. 1988 Feb;26(1):121-8.
The effect of mesterolone on sperm count, on serum follicle stimulating hormone, luteinizing hormone, plasma testosterone and outcome in idiopathic oligospermic men.
Varma TR1, Patel RH.
Author information
Abstract
Two hundred fifty subfertile men with idiopathic oligospermia (count less than 20 million/ml) were treated with mesterolone (100-150 mg/day) for 12 months. Seminal analysis were assayed 3 times and serum follicle stimulating hormone (FSH) luteinizing hormone (LH) and plasma testosterone were assayed once before treatment and repeated at 3, 6, 9 and 12 months after the initiation of treatment. One hundred ten patients (44%) had normal serum FSH, LH and plasma testosterone, 85 patients (34%) had low serum FSH, LH and low plasma testosterone. One hundred seventy-five patients (70%) had moderate oligospermia (count 5 to less than 20 million/ml) and 75 patients (30%) had severe oligospermia (count less than 5 million/ml). Seventy-five moderately oligospermic patients showed significant improvement in the sperm density, total sperm count and motility following mesterolone therapy whereas only 12% showed improvement in the severe oligospermic group. Mesterolone had no depressing effect on low or normal serum FSH and LH levels but had depressing effect on 25% if the levels were elevated. There was no significant adverse effect on testosterone levels or on liver function. One hundred fifteen (46%) pregnancies resulted following the treatment, 9 of 115 (7.8%) aborted and 2 (1.7%) had ectopic pregnancy. Mesterolone was found to be more useful in patients with a sperm count ranging between 5 and 20 million/ml. Those with severe oligospermia (count less than 5 million) do not seem to benefit from this therapy.
PMID: 2892728 [PubMed - indexed for MEDLINE]